أمممم......أنا فين ؟؟؟؟أنا فين؟؟؟؟؟
أمممم أنا عايزة النسخة بتاعتي
أمممم......أنا فين ؟؟؟؟أنا فين؟؟؟؟؟
أمممم أنا عايزة النسخة بتاعتي
حاضر
انا كده هشتغل دليفري بقى
السلام عليكم و رحمة الله و بركاته
دي شوية حاجات للفهم بعد تضيع وقت
في الحالة الأولى
Arterial thrombi are frequently occlusive and are produced by platelet and coagulation activation; they are typically a friable meshwork of platelets, fibrin, erythrocytes, and degenerating leukocytes. Although arterial thrombi are usually superimposed on an atherosclerotic plaque, other vascular injury (vasculitis, trauma) can be involved. Body_ID: PB004013 Venous thrombosis (phlebothrombosis) is almost invariably occlusive, and the thrombus can create a long cast of the lumen; venous thrombosis is largely the result of activation of the coagulation cascade, and platelets play a secondary role. Because these thrombi form in the sluggish venous circulation, they also tend to contain more enmeshed erythrocytes and are therefore called red, or stasis, thrombi. The veins of the lower extremities are most commonly affected (90% of venous thromboses); however, venous thrombi can occur in the upper extremities, periprostatic plexus, or ovarian and periuterine veins; under special circumstances they may be found in the dural sinuses, portal vein, or hepatic vein.
Normal blood flow is laminar, such that platelets flow centrally in the vessel lumen, separated from the endothelium by a slower moving clear zone of plasma. Stasis and turbulence therefore:
Disrupt laminar flow and bring platelets into contact with the endotheliumPrevent dilution of activated clotting factors by fresh-flowing bloodRetard the inflow of clotting factor inhibitors and permit the buildup of thrombiPromote endothelial cell activation, resulting in local thrombosis, leukocyte adhesion, etc
hypercoagulability
It is loosely defined as any alteration of the coagulation pathways that predisposes to thrombosis, and it can be divided into primary (genetic) and secondary (acquired) disorders
العفو يا دندونتي
ربنا يكرمك يااارب
وعليكم السلام ورحمة الله وبركاته....
جزاكِ الله خيــراً يا منى
لما أبدأ مذاكرة بإذن الله هقرأه...
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يا جماعة الخيـــر...
الأسئلة اللي ع الthyroid مش واضحة...
عشان زميلتكم اللي عاملاهم كاتبة بالأخضر
معلش سامحوني
الأسئلة أهي عشان لو حد عايز يتأكد...
1- What symptoms might this mass cause?
2- Compare and contrast clinical features of goiter with those of thyroid neoplasms.
3- What does the abrupt enlargement of the thyroid nodule indicate?
4- What is the biochemical stimulant to hyperplasia in goiter?
5- What is the usual functional outcome of goiter?
6- How would a radioactive iodine scan look in a thyroid gland?
7 -What histologic features would you look for to rule out follicular carcinoma?
8- What is the prognosis for patients with follicular carcinoma?
9- What is the degree of similarity between follicular cell in adenoma & in normal parenchyma
أنا فدائي أنا ...
افتكرت أغنية أنا كويتي أنا ههههه
ربنا يكرمك يا حنين يارب ..تسلمي
I got a permanent ban on the board for being vulgar, reflecting the atmosphere where I am living